| Item Type | Name |
|
Concept
|
Action Potentials
|
|
Concept
|
Air Pressure
|
|
Concept
|
Analgesics, Opioid
|
|
Concept
|
Hypoxia, Brain
|
|
Concept
|
Cerebral Cortex
|
|
Concept
|
Analysis of Variance
|
|
Concept
|
Apnea
|
|
Concept
|
Cell Differentiation
|
|
Concept
|
Cell Nucleus
|
|
Concept
|
Behavior, Animal
|
|
Concept
|
Cadmium
|
|
Concept
|
Biological Clocks
|
|
Concept
|
Biophysics
|
|
Concept
|
Brain
|
|
Concept
|
Brain Stem
|
|
Concept
|
Bromodeoxyuridine
|
|
Concept
|
Central Nervous System
|
|
Concept
|
gamma-Aminobutyric Acid
|
|
Concept
|
Electrophysiology
|
|
Concept
|
Hypoglossal Nerve
|
|
Concept
|
Hippocampus
|
|
Concept
|
Hydrogen Peroxide
|
|
Concept
|
Leukocytes, Mononuclear
|
|
Concept
|
Mammals
|
|
Concept
|
Mice, Transgenic
|
|
Concept
|
Mice, Inbred C57BL
|
|
Concept
|
Respiratory Center
|
|
Concept
|
Synaptic Transmission
|
|
Concept
|
Mitochondria, Heart
|
|
Concept
|
Oxygen
|
|
Concept
|
Polarography
|
|
Concept
|
Periodicity
|
|
Concept
|
Glutamine
|
|
Concept
|
Humans
|
|
Concept
|
Receptors, N-Methyl-D-Aspartate
|
|
Concept
|
Synapses
|
|
Concept
|
Tamoxifen
|
|
Concept
|
Calcium Channels
|
|
Concept
|
Dinoprostone
|
|
Concept
|
Sodium Channels
|
|
Concept
|
Glutamic Acid
|
|
Concept
|
Excitatory Postsynaptic Potentials
|
|
Concept
|
Wakefulness
|
|
Concept
|
Lipid Peroxidation
|
|
Concept
|
Cardiopulmonary Resuscitation
|
|
Concept
|
Morphine
|
|
Concept
|
Rats, Sprague-Dawley
|
|
Concept
|
Medulla Oblongata
|
|
Concept
|
Mice, Knockout
|
|
Concept
|
Excitatory Amino Acid Antagonists
|
|
Concept
|
Cell Proliferation
|
|
Concept
|
Calcium Channels, P-Type
|
|
Concept
|
Receptors, CXCR4
|
|
Concept
|
Calcium Channels, N-Type
|
|
Concept
|
Receptor, Serotonin, 5-HT2A
|
|
Concept
|
Receptors, Adrenergic, alpha-2
|
|
Concept
|
Ryanodine Receptor Calcium Release Channel
|
|
Concept
|
6-Cyano-7-nitroquinoxaline-2,3-dione
|
|
Concept
|
Potassium Channels, Inwardly Rectifying
|
|
Concept
|
Riluzole
|
|
Concept
|
Phrenic Nerve
|
|
Concept
|
Mice
|
|
Concept
|
Rats
|
|
Concept
|
SOXB1 Transcription Factors
|
|
Concept
|
Mice, 129 Strain
|
|
Concept
|
Vertebrates
|
|
Concept
|
Proliferating Cell Nuclear Antigen
|
|
Concept
|
Dentate Gyrus
|
|
Concept
|
Calcium Channels, Q-Type
|
|
Concept
|
Halorhodopsins
|
|
Concept
|
Cell Lineage
|
|
Concept
|
CA1 Region, Hippocampal
|
|
Concept
|
Adrenergic alpha-2 Receptor Antagonists
|
|
Concept
|
Basic Helix-Loop-Helix Transcription Factors
|
|
Concept
|
KATP Channels
|
|
Concept
|
Autonomic Nervous System Diseases
|
|
Concept
|
Calcium
|
|
Concept
|
Calcium Channel Blockers
|
|
Concept
|
Flufenamic Acid
|
|
Concept
|
Norepinephrine
|
|
Concept
|
Receptors, Adrenergic, beta
|
|
Concept
|
Neurogenesis
|
|
Academic Article
|
Point: Medullary pacemaker neurons are essential for both eupnea and gasping in mammals.
|
|
Academic Article
|
Hydrogen peroxide differentially affects activity in the pre-Bötzinger complex and hippocampus.
|
|
Academic Article
|
Chapter 3--networks within networks: the neuronal control of breathing.
|
|
Academic Article
|
A toolbox of Cre-dependent optogenetic transgenic mice for light-induced activation and silencing.
|
|
Academic Article
|
Activation of alpha-2 noradrenergic receptors is critical for the generation of fictive eupnea and fictive gasping inspiratory activities in mammals in vitro.
|
|
Academic Article
|
Graded reductions in oxygenation evoke graded reconfiguration of the isolated respiratory network.
|
|
Academic Article
|
Stable respiratory activity requires both P/Q-type and N-type voltage-gated calcium channels.
|
|
Academic Article
|
Cardiorespiratory coupling in health and disease.
|
|
Academic Article
|
Central and peripheral factors contributing to obstructive sleep apneas.
|
|
Academic Article
|
Prostaglandin E2 differentially modulates the central control of eupnoea, sighs and gasping in mice.
|
|
Academic Article
|
ß-Noradrenergic receptor activation specifically modulates the generation of sighs in vivo and in vitro.
|
|
Academic Article
|
Chronic Intermittent Hypoxia Alters Local Respiratory Circuit Function at the Level of the preBötzinger Complex.
|
|
Academic Article
|
The ins and outs of breathing.
|
|
Academic Article
|
Hyperoxic stimulation of synchronous orthodromic activity and induction of neural plasticity does not require changes in excitatory synaptic transmission.
|
|
Academic Article
|
Neuronal sensitivity to hyperoxia, hypercapnia, and inert gases at hyperbaric pressures.
|
|
Academic Article
|
Hyperbaric hyperoxia and normobaric reoxygenation increase excitability and activate oxygen-induced potentiation in CA1 hippocampal neurons.
|
|
Academic Article
|
The physiological determinants of sudden infant death syndrome.
|
|
Academic Article
|
Keeping carbon dioxide in check.
|
|
Academic Article
|
Different roles for inhibition in the rhythm-generating respiratory network.
|
|
Academic Article
|
Intermittent Hypoxia Disrupts Adult Neurogenesis and Synaptic Plasticity in the Dentate Gyrus.
|
|
Academic Article
|
Respiratory rhythm generation, hypoxia, and oxidative stress-Implications for development.
|
|
Academic Article
|
Neonatal apneic phenotype in a murine congenital central hypoventilation syndrome model is induced through non-cell autonomous developmental mechanisms.
|
|
Academic Article
|
Machine learning-based data analytic approaches for evaluating post-natal mouse respiratory physiological evolution.
|
|
Academic Article
|
Intermittent Hypoxia causes targeted disruption to NMDA receptor dependent synaptic plasticity in area CA1 of the hippocampus.
|
|
Academic Article
|
Developmental defects and impaired network excitability in a cerebral organoid model of KCNJ11 p.V59M-related neonatal diabetes.
|
|
Academic Article
|
Gasotransmitter modulation of hypoglossal motoneuron activity.
|
|
Concept
|
Channelrhodopsins
|
|
Academic Article
|
A consequence of immature breathing induces persistent changes in hippocampal synaptic plasticity and behavior: a role of prooxidant state and NMDA receptor imbalance.
|
|
Academic Article
|
Metabolic trade-offs in Neonatal sepsis triggered by TLR4 and TLR1/2 ligands result in unique dysfunctions in neural breathing circuits.
|